Scientists have found "switches" in the brain that help control appetite, which helps explain why it's hard for some people to know when to stop eating.
Researchers believe that blood sugar levels in the blood are involved in the process of opening this switch in the diet, so people will begin to feel full. Once this switch fails, people will overeating and obesity.
This finding provides further scientific evidence to support George Osborne's new sugar tax. Scientists discovered this appetite switch when studying the strength of the connections between nerve cells in the brain of mice (related to learning and memory).
They especially want to know what happens when they remove the genes that control the synthesis of OGT enzyme in the mouse brain. OGT is involved in multiple metabolisms of the body, including the use of insulin and glucose in the blood. One of the functions of this enzyme is to add a chemical derivative of glucose to the protein. When the scientists deliberately deleted the gene of the enzyme, the body weight of the mouse doubled in just two weeks.
Olof Lagerlof, a researcher at Johns Hopkins University, said: "These mice don't know that they have enough food, so they keep eating." Richard Huganir, head of the Department of Neuroscience at Johns Hopkins University The professor said that after the OGT was no longer working, the mouse did not know when to stop eating. This enzyme controls the balance of the satiety effect. When they increase the activity of OGT enzyme in mice, the hungry normal mice stop eating.
This appetite switch is controlled by the amount of glucose in the blood. After eating or drinking a sugary drink, the blood sugar in the human body will naturally rise.
Professor Huganir said: "It is very likely that human brains have the same mechanisms and brain cell types. We believe that this appetite control switch mechanism may appear in humans, but we still need to do more experiments to confirm this. Theory In fact, if we really understand what happened, then we will be able to use the mechanism of drug human intervention to help people fight the obesity epidemic by controlling people's appetite."
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